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While compelling evidence establishes ferroptosis as a key driver of cyst progression in ADPKDlinked to PKD1/PKD2 mutations, disrupted antioxidant networks (GPX4/SLC7A11), iron overload, and lipid peroxidationand validates ferroptosis modulators as potential therapeutics, several critical, insufficiently addressed research gaps persist
64 The stimulatory effects of ATII on the activity of the Nox would suggest that in the presence of an activated reninangiotensin systema common finding during hypertensionincreased vascular oxidative stress is likely to be expected
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(2010): N-Acetyl-cysteine against noise-induced temporary threshold shift in male workers
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